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LEARNING OBJECTIVES

By the end of this newsletter, you will be able to:

Identify thoracic hypomobility as a contributing factor in shoulder impingement presentations, especially in overhead athletes.

Apply thoracic spine manipulation and mobilization techniques and assess their immediate effect on shoulder range of motion.

Integrate regional interdependence principles into a shoulder rehabilitation program for the swimming athlete.

Evaluate scapular dyskinesis and thoracic mobility using reliable clinical measures and reproducible assessment.

CLINICAL PRESENTATION

L came in referred by her coach, who I trust to spot trouble early. She walked in guarding the right arm slightly across her body. Not dramatic. Just that subtle protective posture you learn to notice.

Subjective

Chief complaint: right shoulder pain, anterior and lateral, worse with overhead activity. Six weeks, gradual onset, no single traumatic event. She points to the front of the shoulder and the outside of the upper arm when I ask her to show me.

The pain is worst during the catch and pull phase of her freestyle stroke, and at the very top of the recovery phase when the arm reaches forward. Night pain when she rolls onto that side. No numbness, no tingling, no neck pain that she notices. She rates it a 6 out of 10 on a bad day, a 3 on a good one.

I ask about training load. She tells me she bumped her yardage in the last two months getting ready for championship season. More volume, more sprint sets, same shoulder doing the same thing thousands of times a day.

"Did anything change with your stroke?" I ask.

"Coach said my catch looked flat. Like I wasn't reaching as far."

That's a clue. Hold onto it.

Objective

Observation: Forward head posture. Rounded shoulders. A flattened, kyphotic-looking upper back, which is extremely common in distance swimmers who live in a flexed, internally rotated position. The right scapula sits slightly more protracted and anteriorly tilted than the left at rest.

Range of Motion: Active shoulder flexion and abduction are limited on the right compared to the left, asymmetrical, with a painful arc in the mid-range of abduction. Internal rotation behind the back is limited on the right. Cervical range of motion is within normal limits and pain free. Here is the finding that matters most: thoracic extension is globally limited and her thoracic rotation is restricted and asymmetrical, more limited toward the right.

Neurological Screening: Myotomes and dermatomes within normal limits. Reflexes symmetrical. No neurological involvement.

Palpation: Tenderness over the anterior and lateral subacromial region. The upper trapezius and levator on the right are tight and tender. The posterior rotator cuff is tender to deep palpation. The thoracic paraspinals from roughly T3 to T7 are hypertonic and restricted, and the costovertebral and costtransverse junctions on the right feel locked.

Orthopedic Tests:

  • Neer impingement test: positive on the right.

  • Hawkins-Kennedy: positive on the right.

  • Painful arc: positive, mid-range abduction.

  • Hawkins on the left: negative.

  • Empty can (Jobe): mildly positive for pain, no significant weakness.

  • Cervical compression (Spurling): negative.

  • Scapular Assistance Test: positive. When I manually assist her scapula into upward rotation and posterior tilt during elevation, her pain decreases and her range improves.

Scapular Assessment: Visible scapular dyskinesis on the right during arm elevation and lowering. The medial border and inferior angle wing slightly, and the scapula fails to upwardly rotate and posteriorly tilt the way it should as she raises her arm. The Scapular Assistance Test is the tell. When I correct the scapular motion by hand, her symptoms change immediately.

So what's the picture here?

Three previous providers stopped at the shoulder. They saw positive Neer, positive Hawkins, anterior shoulder pain, and they called it impingement. That is not wrong. It is just not the whole story.

This is not a primary rotator cuff problem. This is subacromial impingement driven by scapular dyskinesis, and the scapular dyskinesis is being driven by a stiff, hypomobile thoracic spine. The thoracic spine cannot extend or rotate, so the scapula cannot position itself correctly, so the subacromial space narrows every time she reaches overhead. Thousands of times a day.

Time to fix it. But first, let me show you why so many providers miss this.

COMMON MISDIAGNOSIS

The Trap: Diagnosing isolated rotator cuff pathology or "swimmer's shoulder" and treating only the shoulder, while the thoracic spine that is actually driving the problem goes completely unexamined.

Here is what happens in most clinics. The athlete comes in with anterior shoulder pain and a positive impingement sign. The provider runs the shoulder special tests, gets a positive Neer and Hawkins, and lands on subacromial impingement or rotator cuff tendinopathy. The treatment plan writes itself: rotator cuff strengthening, maybe some scapular exercises, ice, activity modification, rest.

And the athlete does not get better. Or they get a little better, then plateau, then flare the moment they return to full training.

The reasoning error is not the diagnosis of impingement. The error is treating impingement as a local problem. Impingement is a sign, not a root cause. The subacromial space narrowed for a reason. If you do not ask why, you treat the symptom and miss the driver.

Why This Happens:

  • Special tests point local. Neer and Hawkins reproduce subacromial pain, so the clinician's attention stays glued to the shoulder. The tests are doing their job. The clinician just stops looking too soon.

  • The thoracic spine is rarely assessed in a shoulder exam. Most shoulder evaluations never include thoracic extension or rotation testing. If you do not look there, you cannot find it there.

  • Regional interdependence is taught but not applied. Almost every clinician has heard the phrase. Far fewer actually screen up and down the kinetic chain when the chief complaint is at one joint.

  • Swimmers get a label. "Swimmer's shoulder" is a wastebasket term that lets everyone stop thinking. It describes a population, not a diagnosis.

The Cost:

For L, the cost was six weeks of pain, declining performance, and a flattening catch that her coach already noticed. The stroke change is the body protecting itself. She was shortening her reach to avoid the painful position, which kills her distance per stroke, which kills her times right before championship season.

The bigger cost is the trajectory. A swimmer who keeps training through scapular dyskinesis and thoracic stiffness does not stay at a minor impingement. The repeated mechanical irritation can progress to rotator cuff tendinopathy, partial thickness tearing, and chronic pain that follows them out of the sport. Three providers treated the shoulder. None of them touched the thoracic spine. That is six weeks she will not get back, and a stroke she now has to rebuild.

This is fixable. But you have to look one region down.

CORRECT DIFFERENTIAL DIAGNOSIS

Subacromial Impingement (scapular driven) vs. Primary Rotator Cuff Tendinopathy vs. Cervical Referral

The job here is to separate three things that all produce anterior and lateral shoulder pain in an overhead athlete. Is this pain coming from the subacromial structures because the scapula is mispositioned? Is it primary tendon pathology in the cuff itself? Or is it referred from the cervical spine? The tests sort it out.

Diagnostic Tests Performed:

Neer Impingement Test: Positive Finding: Pain reproduced with passive overhead elevation on the right. Clinical Significance: Confirms subacromial structures are being compressed. Tells you the space is the problem, not where the problem starts.

Hawkins-Kennedy Test: Positive Finding: Pain with the provocative internally rotated position on the right. Clinical Significance: Reinforces subacromial involvement. Combined with Neer, the subacromial space is clearly implicated.

Scapular Assistance Test: Positive Finding: Manual assistance of scapular upward rotation and posterior tilt reduced pain and improved elevation. Clinical Significance: This is the key test. A positive result tells you the scapula is a primary contributor and that improving scapular mechanics will improve symptoms. This shifts the diagnosis from local cuff problem to scapular dyskinesis driven impingement.

Empty Can (Jobe) Test: Mildly positive for pain, no weakness Clinical Significance: Some supraspinatus irritation, but the absence of true weakness argues against a significant cuff tear and against primary tendinopathy as the main driver.

Spurling (Cervical Compression) Test: Negative Finding: No reproduction of shoulder or arm symptoms. Clinical Significance: Rules out cervical radicular referral as the source. The neck is not driving this.

Thoracic Mobility Assessment: Positive for restriction Finding: Limited and asymmetrical thoracic extension and rotation, more restricted on the right, with hypomobile T3 to T7 segments and locked right costovertebral junctions. Clinical Significance: This is the upstream driver. A stiff thoracic spine prevents the scapula from achieving the upward rotation and posterior tilt needed to clear the subacromial space during elevation.

DECISION FRAMEWORK:

CLINICAL FINDINGS

MOST LIKELY DIAGNOSIS

CONFIDENCE LEVEL

NEXT STEPS

Positive Neer/Hawkins, positive Scapular Assistance Test, restricted thoracic mobility

Scapular dyskinesis driven subacromial impingement

High

Treat the thoracic spine and scapular mechanics first, reassess shoulder

Positive Neer/Hawkins, negative Scapular Assistance Test, significant cuff weakness

Primary rotator cuff tendinopathy or tear

Moderate to High

Progressive cuff loading, consider imaging if weakness persists

Shoulder pain reproduced with Spurling, neurological signs present

Cervical radicular referral

Moderate

Cervical workup, do not chase the shoulder

Mixed picture: positive impingement signs, partial scapular response, some cuff irritation

Multifactorial impingement with both scapular and cuff components

Moderate

Address thoracic and scapular drivers, then load the cuff progressively

Night pain, progressive weakness, loss of active range with preserved passive range

Possible full thickness cuff tear or serious pathology

Lower for benign, requires rule out

Refer for imaging and orthopedic consult

The decision framework is the part of this newsletter readers screenshot. Keep it next to your shoulder exam.

REFERRAL CRITERIA (When to Send Out)

Immediate Emergency Referral:

  • Signs of acute neurovascular compromise in the arm, including loss of pulse, cold or pale limb, or rapidly progressing numbness.

  • Suspected fracture or dislocation after acute trauma with deformity.

  • Sudden, severe, unremitting pain with constitutional symptoms such as fever or unexplained weight loss.

  • Any sign of an evolving neurological deficit not explained by the musculoskeletal exam.

Urgent Medical Referral (Same Day):

  • Significant active weakness suggesting a large or full thickness rotator cuff tear, especially after trauma.

  • Suspected acute infection or systemic illness involving the joint.

  • New, unexplained night pain that is severe and not positional.

Co-Management Referral:

  • Failure to progress after a reasonable course of conservative care, where an orthopedic or sports medicine opinion would guide next steps.

  • Suspected labral involvement or instability that may need imaging and a surgical opinion. I continue manual care and rehabilitation alongside the specialist.

  • The young athlete heading into a high stakes competition season where shared decision making with the medical team protects the athlete.

Imaging Referral:

  • Persistent weakness or loss of active range despite conservative care, where ultrasound or MRI clarifies cuff integrity.

  • Suspected structural pathology of the labrum or biceps anchor not responding to treatment.

  • Atypical presentation that does not fit a mechanical pattern, to rule out serious pathology.

L did not meet any of these criteria. Clean neuro screen, no significant weakness, no red flags. A mechanical problem with a mechanical solution.

MANUAL THERAPY: THORACIC MANIPULATION AND MOBILIZATION

Here is where the evidence and the clinical reasoning come together.

The whole case turns on regional interdependence. The thoracic spine and the shoulder are not separate systems. When the thoracic spine cannot extend and rotate, the scapula cannot position correctly on the rib cage, and the subacromial space narrows during elevation. Free up the thoracic spine, and the scapula gets the room it needs to move.

This is not theory. According to PubMed, a 2025 systematic review and meta-analysis in the Archives of Physical Medicine and Rehabilitation pooled 10 randomized controlled trials and found high quality evidence that thoracic spine manual therapy produces a large effect on pain in subacromial impingement at short term follow up, with significant improvements in disability and moderate evidence for improved shoulder rotation. That matches exactly what I see in the clinic.

And we can be specific about the mechanism. A 2017 randomized controlled trial, also in the Archives of Physical Medicine and Rehabilitation, showed that thoracic spine manipulation increased scapular upward rotation during arm lowering in patients with shoulder impingement. Upward rotation is precisely the motion L is missing. Her Scapular Assistance Test told me the same thing before I read a single study.

There is even imaging evidence for the space itself. A 2018 study in the Journal of Manipulative and Physiological Therapeutics measured the subacromial space with ultrasound and found it increased significantly after thoracic spine manipulation in neutral and external rotation. So the manipulation does not just feel better. The space the shoulder needs actually opens up.

Here is exactly what I did.

Thoracic Spine Manipulation:

For the hypomobile mid thoracic segments, T3 through T7, I used a supine anterior to posterior thrust manipulation. Patient supine, arms crossed, I roll her toward me, position my hand under the target segment, and deliver a controlled, high velocity, low amplitude thrust through the crossed arms on exhalation. I felt and heard cavitation at the restricted segments.

For the rotational restriction, I used a seated thoracic rotation manipulation to restore the asymmetrical rotation, focusing on the right sided restriction that mirrored her shoulder limitation.

Expected response: immediate improvement in thoracic extension and rotation, and critically, immediate improvement in pain free shoulder elevation when you reassess right after. If the shoulder does not change after you free the thoracic spine, you targeted the wrong driver. Reassess.

Thoracic and Costovertebral Mobilization:

For the locked right costovertebral and costotransverse junctions, and for segments where thrust was not appropriate, I used graded posterior to anterior mobilizations in prone. Grade III and IV oscillations at the stiff segments. This is also the technique I teach for athletes who are apprehensive about thrust manipulation, and for the days between manipulation sessions.

Mobilization is not the lesser option. The critically appraised topic published in 2025 in the Journal of Sport Rehabilitation concluded that adding either thoracic manipulation or mobilization to exercise produced greater improvements in shoulder range of motion, pain, and disability than exercise alone. Manipulation and mobilization are both legitimate tools. Pick based on the segment, the patient, and the response.

Immediate Reassessment:

This is the non negotiable step. After manipulation and mobilization, I reassessed her thoracic extension, her shoulder elevation, and the painful arc. Her overhead elevation improved immediately and the painful arc diminished. That immediate change confirms the diagnosis and earns the athlete's buy in. She felt it move.

One honest note on the evidence. Not every study agrees. A couple of systematic reviews report that thoracic manipulation as a standalone treatment shows inconsistent results, and that the clearest, most durable gains come when manipulation is paired with exercise. I agree completely. Manipulation opens the door. Rehabilitation walks her through it and keeps it open. Manual therapy alone is a short term win. We need both.

THE CDNP APPROACH

The thoracic spine was the driver, but L also walked in with genuinely angry tissue. Six weeks of guarding gave her a tight, tender upper trapezius and a posterior cuff that did not want to let go. Dry needling here is not about chasing the pain. It is about quieting the protective muscle tone so the new thoracic and scapular mechanics can actually be used.

Clinical reasoning: when you restore thoracic mobility but the periscapular muscles are still locked in a guarded, facilitated state, the scapula cannot move into its new available range. Needling the right muscles releases that guard and lets the rehabilitation take hold faster.

TARGET MUSCLES:

Upper Trapezius (Unilateral, right) Anatomical Landmarks: The muscle belly between the cervical spine and the acromion, needled into the anterior fibers with a pincer grip to keep the needle away from the apex of the lung. Needle Specifications: 0.25mm x 30mm Depth: Approximately 10 to 15mm into the pincered muscle belly. Note: Depth is patient dependent based on body composition, muscle development, and tissue response. Always assess tissue resistance and patient feedback to determine appropriate depth for each individual. Technique: Pincer grip, gentle pistoning to elicit a local twitch response. Expected Response: Local twitch, then a softening of the muscle tone. The patient often reports the shoulder feels lighter. Safety Notes: The pincer grip is essential here. Needle into the tented tissue, never flat toward the lung. This is the single most important pneumothorax precaution in the upper trapezius.

Infraspinatus (Unilateral, right) Anatomical Landmarks: Over the body of the scapula, below the spine of the scapula, into the muscle belly against the solid backstop of the scapular bone. Needle Specifications: 0.30mm x 40mm Depth: Approximately 15 to 25mm, advancing onto the flat bone of the scapula. Note: Depth is patient dependent based on body composition, muscle development, and tissue response. Always assess tissue resistance and patient feedback to determine appropriate depth for each individual. Technique: Pistoning to the scapular backstop, seeking twitch in the trigger points. Expected Response: Strong local twitch, reproduction of the patient's familiar posterior ache, then release. Safety Notes: The scapula is your safety floor. Stay over the bone of the scapular body and you stay safe. Be cautious near the medial and inferior borders where you can move off bone.

Levator Scapulae (Unilateral, right) Anatomical Landmarks: The superior medial angle of the scapula where the levator inserts. Needle Specifications: 0.25mm x 30mm Depth: Approximately 10 to 15mm toward the superior angle of the scapula. Note: Depth is patient dependent based on body composition, muscle development, and tissue response. Always assess tissue resistance and patient feedback to determine appropriate depth for each individual. Technique: Direct toward the superior angle, gentle pistoning. Expected Response: Twitch and release at the insertion, relief of the upper medial scapular tension. Safety Notes: Angle the needle toward the scapula at the superior angle. Stay lateral to the safe zone and respect the rib cage and lung below the medial border.

TREATMENT FREQUENCY: Phase 1 (Initial, Week 1): One to two sessions, paired with the first manipulation, to break the guarding cycle quickly while it is most active. Phase 2 (Stabilization, Weeks 2-3): Once per week as needed, only the muscles that are still holding tone. As mechanics improve, the need for needling drops fast. Phase 3 (Maintenance, Weeks 4+): As needed only. By this phase, the goal is active control through rehabilitation, not passive release.

REHABILITATION

Manipulation opens the window. Rehabilitation is what keeps it open. This is where regional interdependence becomes a daily practice rather than a single treatment.

The plan has to do three things. Maintain the thoracic mobility we just restored. Retrain the scapula to upwardly rotate and posteriorly tilt during elevation. And rebuild the swimming specific endurance that distance freestyle demands. The evidence is clear that the combination of manual therapy and exercise beats either alone, so the rehab is not optional. It is the half of the treatment that lasts.

REHABILITATION SAFETY PRINCIPLES: Train in the pain free range first, then expand it. Swimmers are tough and will push into pain if you let them. Quality of scapular motion comes before load and before volume. Keep early loading below the painful arc until mechanics clean up.

Phase 1, Foundation (Weeks 1-2):

  • Thoracic extension over a foam roller, segmental, 2 sets of 10 controlled extensions, daily. This maintains what the manipulation restored.

  • Open book thoracic rotation in side lying, 2 sets of 8 per side, daily, emphasizing the restricted right rotation.

  • Scapular setting and low row holds, teaching posterior tilt and upward rotation, 3 sets of 10 second holds.

  • Serratus anterior activation with a wall slide, focusing on protraction and upward rotation, 3 sets of 10.

Phase 2, Loading (Weeks 3-4):

  • Prone Y, T, and W raises for lower and middle trapezius, 3 sets of 12, controlled, no shrugging.

  • Serratus punch progression with light resistance, 3 sets of 12.

  • Banded external rotation at the side, building posterior cuff endurance, 3 sets of 15.

  • Quadruped thoracic rotation reaches, integrating thoracic mobility with scapular control, 2 sets of 10 per side.

Phase 3, Sport-Specific Integration (Weeks 5+):

  • Standing banded freestyle catch and pull simulation, training the scapula to position correctly through the stroke pattern, 3 sets of 12.

  • Prone swimmer's press and scapular endurance circuits that mirror the demands of distance freestyle.

  • Closed chain and weight bearing scapular control drills for the recovery phase position.

  • Gradual return to yardage, starting at reduced volume with drill focus on a long, full catch, then building distance as mechanics hold.

RETURN-TO-SPORT CRITERIA:

  • Full, symmetrical, pain free overhead range with a clean painful arc.

  • Negative or markedly improved Scapular Assistance Test, with the scapula upwardly rotating without manual help.

  • Symmetrical thoracic extension and rotation maintained between sessions.

  • Ability to complete sport specific scapular endurance work without fatigue induced dyskinesis.

  • A return to full yardage built in stages, with the coach monitoring the catch for the flattening that started this whole thing.

ETHICAL CONSIDERATIONS

Treating the Minor Athlete Under Competitive Pressure

L is 17, heading into championship season, and there is real pressure in the water around her. The coach wants her ready. The athlete wants to compete. The parents want her healthy. These goals usually align, but not always. My job is to make the clinical decision based on the tissue in front of me, not the meet schedule on the wall. If returning to full yardage too soon risks turning a reversible impingement into a chronic cuff problem, I have to say so plainly, even when the timing is inconvenient. The athlete's long term shoulder outweighs any single season. With a minor, that responsibility is heavier, not lighter.

Honest Communication About Manual Therapy's Limits

When a patient feels their shoulder move better immediately after manipulation, it is powerful, and it can be oversold. The honest framing is that manipulation produces a meaningful short term improvement and that the durable result depends on the rehabilitation she does on her own. I owe her the truth that the table work is the easy part and the homework is the part that lasts. Overselling the passive treatment quietly undermines the active one, and the active one is what protects her shoulder for the next decade.

Coordinating Care When Other Providers Were Involved

Three providers saw L before me. It would be easy, and wrong, to run them down to make myself look sharp. The professional move is to explain the reasoning, not to disparage colleagues. Impingement was a reasonable read. The thoracic driver is simply a layer that is easy to miss without screening for it. I keep the door open for co management and I document my findings clearly enough that the next provider in the chain can follow the logic. Respect for the rest of the care team serves the athlete better than ego ever will.

CLINICAL PEARLS

  1. When a shoulder impingement will not resolve, screen the thoracic spine before you blame the cuff. The driver is often one region down.

  2. The Scapular Assistance Test is your fastest diagnostic shortcut. If correcting scapular motion by hand changes the symptoms, the scapula is a primary player and the thoracic spine is your next stop.

  3. Always reassess shoulder elevation immediately after thoracic manipulation. The immediate change confirms your diagnosis and earns the athlete's trust in one move.

  4. Manipulation opens the window, rehabilitation keeps it open. The evidence is strongest when you combine manual therapy with exercise, so never send the athlete home with table work alone.

  5. In distance swimmers, a flattening catch is an early warning sign. The body shortens the reach to dodge the painful position long before the athlete complains.

  6. Needle to quiet the guard, not to chase the pain. Releasing protective periscapular tone lets the restored mechanics actually get used.

  7. Forward head, rounded shoulders, and a flat upper back in an overhead athlete is a thoracic problem waiting to become a shoulder problem. Treat the posture before it becomes a diagnosis.

SOAP NOTE TEMPLATE

Subjective: 17 year old female competitive distance swimmer presents with six week history of insidious onset right shoulder pain, anterior and lateral, rated 3 to 6 out of 10. Pain is worst during the catch and pull and at the top of the recovery phase of freestyle, with positional night pain on the right side. Reports recent increase in training yardage and a coach noted flattening of the stroke catch. No numbness, tingling, or neck pain. Three prior providers diagnosed rotator cuff involvement or swimmer's shoulder with limited improvement.

Objective: Posture: forward head, rounded shoulders, flattened thoracic kyphosis, right scapula resting in protraction and anterior tilt. ROM: right shoulder flexion and abduction limited and asymmetrical compared to left with a mid range painful arc, right internal rotation limited. Cervical ROM within normal limits and pain free. Thoracic extension globally limited, thoracic rotation restricted and asymmetrical, greater on the right. Neuro: myotomes and dermatomes within normal limits, reflexes symmetrical. Palpation: tenderness over the subacromial region, tight and tender right upper trapezius, levator, and posterior cuff, hypomobile T3 to T7 segments, locked right costovertebral junctions. Special tests: Neer positive right, Hawkins-Kennedy positive right, painful arc positive, Scapular Assistance Test positive, Jobe mildly positive for pain without weakness, Spurling negative. Visible scapular dyskinesis on right with elevation and lowering.

Assessment: Right subacromial impingement secondary to scapular dyskinesis, driven by thoracic spine hypomobility, in a distance swimmer with increased training load. Positive Scapular Assistance Test and restricted thoracic mobility support a regional interdependence model rather than primary rotator cuff pathology. Negative Spurling rules out cervical referral. Absence of significant weakness argues against full thickness cuff tear. No red flags requiring urgent referral.

CASE RESOLUTION

L responded the way these cases tend to respond when you treat the actual driver.

After the first session of thoracic manipulation, costovertebral mobilization, and dry needling to quiet the guarding, her overhead elevation improved on the spot and the painful arc diminished. She felt the difference before she left the table, which made her a believer in the homework, which is exactly what I needed. By the end of the second week, with consistent thoracic mobility work and scapular retraining, her night pain was gone and her resting pain had dropped to an occasional 1 out of 10.

By week four she was back to building yardage in stages, leading with drill work on a long, full catch. Her coach reported that the stroke was reaching again, the flattening was gone, and her distance per stroke was back. We cleared her for full training ahead of championship season, with the scapular endurance circuits folded into her dryland routine to keep the mechanics honest under fatigue.

Here is the lesson I want you to carry out of this one. The shoulder is where it hurt, but the shoulder was not the problem. The thoracic spine could not give the scapula room to move, so the subacromial space paid the price thousands of times a day. Three providers treated where it hurt. The fix was one region down.

Screen the thoracic spine on every stubborn shoulder. Reassess after you manipulate. Pair the manual work with the rehab. And remember that every athlete presents a little differently, so the protocol points you in a direction while your hands and your reasoning fill in the rest. That is the work. That is what makes it worth doing.

See you in the next issue.

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In health and strength,

Dr. Thomas Kauffman, DC, CDNP, CSCS, USAW
The Clinical Coach™

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